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- Fernando A. Castro, V
- Resident, Large Animal Surgery
- UTCVM - LACS
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- “If your horse is lame in his shoulder, take off his shoes… Young and
inexperienced practitioners are quite too apt to commit the error of
overlooking the examination of the foot, looking upon it as a matter of
secondary importance, and attending to it as a routine and formal affair
only.”
- A. Liautard, 1888.
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- Lame(adj.): crippled or physically disabled,as a person or animal,… in
the foot or leg so as to limp or walk with difficulty.
- Lameness: incapable of normal locomotion, deviation from the normal
gait.
- Claudication: limping or lameness.
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- “Lameness is therefore not so much an original evil, a disease per se,
as it is a symptom and manifestation of some antecedent vital physical
lesion, either isolated or complicated, affecting one or several parts of the locomotive
apparatus.”
- Liautard, 1888.
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- History, physical exam, manipulative tests, joint and nerve blocks,
diagnostic imaging.
- Characteristic gait abnormalities (sweeny, fibrotic myopathy, upward
fixation of patella, stringhalt, etc.).
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- Baseline x induced lameness
- Manipulative tests
- Coexistent lameness (compensatory or secondary).
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- Computed Assisted Tomography
- Magnetic Resonance Imaging
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- Arthritis/arthrosis (degenerative joint disease, DJD).
- Navicular disease (caudal heel pain).
- Laminitis
- Severe soft tissue injuries.
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- Causes and predisposing factors:
conformation, trauma (repetitive
impact or acute trauma), overload, etc.
- Clinical signs: joint distension, pain, heat, reduced range of motion
(stiffness).
- Affected joints: knee, fetlock, hock (bone spavin), pastern (ring bone),
stifle, etc.
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- Clinical signs
- Lameness exam
- Diagnostic imaging
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- Non-steroidal anti-inflammatories.
- Intra-articular corticosteroids
and hyaluronic acid.
- Systemic hyaluronic acid and glucosaminoglycans (Legend, Adequan).
- Corrective trimming & shoeing.
- Exercise
- Nutraceuticals (glucosamine & chondroitin)
- Physical therapy and alternative therapies.
- Arthrodesis
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- What is it?
- First reported 230 years ago.
- Responsible for approx. 1/3 of chronic lameness.
- Navicular apparatus.
- Cause(s): ????
- Multifactorial: conformation, stress
and non-adaptative remodeling, hereditary.
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- Bilateral forelimb lameness (one side worse).
- Age: 7-9 years.
- Quarter Horse, Warmblood and
Thoroughbred cross.
- “Choppy” gait (short stride). Worse on hard surfaces and when circling.
- Worse with rest.
- Hoof imbalances.
- “Pointing” of the feet.
- Hoof testers: +/-
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- History and clinical signs.
- Lameness exam and nerve blocks (intra-articular and intra-bursal
blocks).
- Diagnostic imaging (radiography,
scintigraphy – “bone scan”, MRI).
- Response to treatment and trimming/shoeing changes.
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- Corrective trimming and shoeing. (shorten toe, preserve heel, maintain
balance of hoof, elevate heels,
- Non-steroidal anti-inflammatories + exercise.
- Intra-articular (coffin joint) or intra-bursal injections.
- Nutraceuticals.
- Isoxsuprine, pentoxyfiline, warfarin.
- Neurectomy (“nerving”).
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- What is laminitis?
- Anatomy of the hoof: tough, flexible, connective tissue apparatus
suspends the coffin bone inside the hoof wall.
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- Pain (stiff gait, shifting weight to rear legs) of acute onset, usually
on both front but can occur on
all four or one limb only.
- Lameness accentuated as animal turns or on hard surface.
- Increase in digital pulses, warm hooves.
- Clinical signs are usually diagnostic.
- Usually secondary to a disease process (colic, retained placenta,
Cushing’s disease, etc).
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- Treat primary problem (iv fluids, uterine lavage, pergolide – Cushing’s,
etc).
- Anti-inflammatories, hoof support (Styrofoam, dental impression
material, plaster of Paris, Lily pads).
- Cold therapy during developmental phase.
- Vasodilators (acepromazine, Isoxsuprine, glyceryl trinitrate patches)
when clinical signs are evident.
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- Radiographic monitoring +/- venogram.
- Trimming & shoeing (very important!!!).
- Deep digital flexor tenotomy.
- Prognosis and management.
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